Advances in Critical Care Testing: The 1996 IFCC-AVL Award by T. Suzuki (auth.), Univ.-Prof. Dr. med. W. F. List, M. M.

By T. Suzuki (auth.), Univ.-Prof. Dr. med. W. F. List, M. M. Müller, M. J. McQueen (eds.)

This quantity on clinical advances in severe care trying out com­ piles a few medical and laboratory stories with regards to criti­ cally unwell sufferers that contain new expertise, remedy recommendations, program or interpretation of latest checks, sufferer administration and price advantages. there have been a complete of 340 candidates for this primary foreign Federation of scientific Chemistry-AVL Award, and from those, nationwide Winners have been chosen in 26 member international locations of the IFCC. This ebook offers the complete papers of the 10 finalists one of the nationwide Winners chosen for the foreign ultimate held in London in July 1996. those ten have been selected by way of the Inter­ nationwide Awards Committee of the IFCC. moreover, the editors have made up our minds additionally to incorporate their collection of the simplest abstracts from the nationwide Winners, therefore giving a vast evaluation of cur­ lease study being performed within the box of severe care medi­ cine between IFCC participants. in accordance with the identify of this quantity, all significant fields of in­ tensive care drugs are represented, together with irritation, an infection, rigidity, hypoxia, ischaemia, cardiology, haemodynamics, blood gases, electrolytes, hint -elements, nephrology, gastroenter­ ology and haematology. moreover, there's additionally a bankruptcy on new expertise in severe care trying out and on miscellaneous most sensible­ ics.

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However, elastases are thought to contribute to some forms of lung injury [11], and instillation of chemotactic pep tides has long been known to promote an inflammatory reaction in the lung [12]. Furthermore, exposure to superoxide dismutase promotes pulmonary inflammation [13], and oxygen metabolites have been shown to damage the lungs of laboratory animals [14]. It is possible that the addition of gram-negative bacilli to this mixture, and particularly their lipopolysaccharides, might augment the toxic effect of these host-derived inflammatory mediators.

Losser and D. Payen Discussion To our knowledge, no study has evaluated the impact of glucose availability simultaneously on the haemodynamic, metabolic and inflammatory response to EDTX. According to the literature, modification of glucose metabolism after EDTX challenge seems to have haemodynamic [8] or immune consequences [17, 19]. Because of the long surgical preparation before EDTX injection, control values may have been influenced by surgical stress. This non-specific inflammation may account for the detectable plasma TNF-a and for the elevated lactate levels without metabolic acidosis or hypoxia seen in fed animals.

2). 05). Glucose and lactate variations significantly differed from the fed animals (Fig. la). 6000 . , . - _ - . L--r--'-'T TO -l Fig. 3. Arterial kinetics of plasma tumour necrosis factor (TNF-a) in endotoxin (EDTX)-treated rabbits in fed (white bars), fasting (shaded bars) and glucose-loaded, fasting (black bars) groups. 05 for intergroup comparison, ~ for intragroup comparison, two-way analysis of variance (ANOVA) for repeated measures, data expressed as mean±SE Glucose Metabolism Modifies Experimental Endotoxin Shock 37 In glucose-loaded, fasting animals, EDTX challenge induced different patterns when compared with the other two groups.

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